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My speculation is Mason is another disinformation agent deflecting from the real culprit of rising diseases - THE INJECTIONS.

It is only speculation on my part, but based on the sheer volume and financial gains of agents pursuing their true craft.

Thank you so much for your intensive truthful work. I knew the entire "seed oil is deadly" thing was too coincidentally spread far and wide around 3 to 4 years ago.

Also, gotta love that name - "Mason". God Bless.

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I had never even considered that possibility, but now that you've raised it, I find it intriguing. I just looked up "seed oils" on Google Trends (https://trends.google.com/trends/explore?date=today%205-y&geo=AU&q=%22seed%20oils%22&hl=en-AU). There were a few spikes in searches in 2019 and 2020, but searches exploded in 2022, coinciding with the booster roll-outs. This could be a purely organic phenomenon, of course, driven by increased traction that the low-carb/carnivore influencers have been getting in recent years. But as you and I both know, that traction is heavily influenced by algorithmic boosting.

I noticed that a lot of the early dissent to COVID tyranny emanated from the keto/paleo/carnivore crowd, and I found that very interesting. I assumed it was because they already had an anti-authoritarian bent due to their distrust of 'expert' nutritional advice (which isn't entirely unwarranted, of course). But if my memory of those times is accurate, very little of that convid-questioning content was censored. Hmmmm. Almost as if they were permitted to stay online in order to serve some wider purpose.

You're spot on about these people making big bucks out of their 'influencing'. And they do it on platforms which are fully on board with the WEF 'meat is killing the earth/eat ze bugs' agenda. Those platforms have no qualms about shutting down other content creators with unapproved messages, yet they permit these messages to spread. It's a head-scratcher. Yet another divide-and-conquer play, perhaps?

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There have been so many deflections used to point away from any source of catastrophic decline in health other than the injections.

Shoveling snow. Being too happy. A myriad of lifestyle choices especially dietary.

Whenever I see something being pushed at the same time something else is being hidden, I wonder what's up.

This started for me over ten years ago when a study from Europe was published regarding cancer and tobacco and why Americans have such a high rate of tobacco related cancers when Europeans do not yet Europeans smoke a great deal. Of course it was discovered the chemicals found in American tobacco were the real culprits. Chemicals that are banned in European tobacco.

But several unintended results also came from the studies. The most interesting being that nicotine is one of the only substances that blocks fluoride uptake in the brain and that the anti-smoking campaign in America began at the same time wide-spread fluoridation of municipal water supplies began.

Now, nicotine has been prescribed to patients suffering from "long covid" to block the uptake of "spike proteins" and has helped.

There are more twisted agendas more easily spread with little effort due to social media and the internet in general.

Seed Oils Bad is looking like one to me.

With AI as trained as it has become, many nuanced agendas and prep for future psyops can be simultaneously organized and implemented with less and less efforts and in record time. This is psychological warfare and the sort most people fail to realize even exists. Most unfortunately.

I remember when CV19 was first launched, people around me were saying they wouldn't drink bleach to prevent it and couldn't believe people were doing such a thing. There were memes all over social media of Trump drinking from a bleach bottle, etc. Of course, what they were really talking about is Chlorine Dioxide Solution (CDS) which has benefited many people with many illnesses for many years with few side effects and is not bleach, but the name sounds like bleach. Psyop.

Again, thank you for all your work.

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I have two clients who are using nicotine (one in the form of patches, the other as chewing gum) to help with conditions they developed after exposure to the convid jabs (one directly, the other via 'shedding'). The results they've had are quite impressive.

The 'Trump recommended drinking bleach to fight COVID' meme was absolutely a psyop. Even Politifact (https://www.politifact.com/factchecks/2020/jul/11/joe-biden/no-trump-didnt-tell-americans-infected-coronavirus/) concedes that Trump discussed the possibility of using UV light, which has a disinfecting effect, as a treatment for COVID. Yet all the lamestream lapdog media maniacally parrotted the claim that Trump had recommended drinking bleach. Utterly disgusting behaviour.

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The people who engaged in this disgusting behavior have blood on their hands.

I know that's blunt, but it's how I see it. Free will and the consequences.

It is wonderful you're able to help people. I have Morgellons/CDB and I understand how valuable medical help can be or the lack thereof not be.

God Bless you.

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You wanted data that correlated what appears to be IR.

Roughly 15% increase in PUFA seems to cause a 15 point increase in bs, with no change in Insulin level. To me this looks like it. Clearly this a select group of individuals, but I doubt it's uniquely isolated. I'm surprised you have a question.

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The authors themselves stated that "we did not observe any change in insulin, HOMA, ISI, adiponectin, or TNFα". HOMA (Homeostatic Model Assessment for Insulin Resistance) is the most widely accepted test for insulin resistance and ISI (insulin sensitivity index) is also widely used. Adiponectin is a fat-derived hormone that plays a crucial role in protecting against insulin resistance. TNF-α induces insulin resistance in adipocytes and peripheral tissues by impairing insulin signaling. To put it plainly, the study did not find ANY EVIDENCE WHATSOEVER that the walnut oil used in this study (NOT a "seed oil") induced insulin resistance. Did you actually read this study?

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Here is an excerpt.

"The PUFA-rich diet increased plasma linoleic acid from 28.36 ± 1.00% to 33.76 ± 1.08% (P < 0.002) and α-linolenic acid from 0.52 ± 0.03% to 1.06 ± 0.10% (P < 0.0001). Fasting glucose increased from 76 ± 3 to 95 ± 3 mg/dl (4.2 ± 0.2 to 5.30.2 mmol/liter; P < 0.0001), and the area under the curve for glucose during oral glucose tolerance test increased from 421 ± 34 to 503 ± 31 mg/dl (23.4 ± 1.9 to 27.9 ± 1.7 mmol/liter; P < 0.001). Plasma insulin did not change either at fasting or during oral glucose tolerance test. "

Your argument is insulin didn't change, if you notice, that's what I said. What did change is both the fasting glucose and the glucose in the glucose tolerance test. This is all based on a modest input change in Linoleic and linolenic, main features of seed oils. Walnut is a seed, just not the more common ones, and I'd consider it better than soybean oil.

If Insulin levels were the brake pressure on a vehicle speed and the vehicle was moving 15% faster, I would say the brakes aren't working as well, even if the car weighs less.

Here is some additional data which could not be so easily compiled from human physiology due to testing methods.

This study compares rates of Linoleic at 1% vs 8% on Low Fat and Medium Fat diets. It was a follow up to a similar 1% and 8% Medium fat and a high fat diet which also included EPA and DHA for comparison. You will also see imagery of the the size of the fat cells. The 1% diet would be something that approximated pre industrial eating. The 8% was approximated current eating. The human study above, was only 6% prior to the addition of the walnut oil, featuring linoleic oil.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3889814/pdf/11745_2013_Article_3842.pdf

https://onlinelibrary.wiley.com/doi/epdf/10.1038/oby.2012.38

After awhile. this led to finding someone who had reviewed these as well as significantly more. I'm including his more comprehensive work.

He notes the association between Linoleic acid association with increased obesity, as well as with it being associated with the drive to eat more, which apparently is activated via the vagus nerve attachment in the stomach. Speaks of efforts to interrupt this signal. and more. At the end of the second part he describes 4-Hydroxynonenal (HNE), which induces obesity.

https://tuckergoodrich.substack.com/p/does-linoleic-acid-induce-obesity

https://tuckergoodrich.substack.com/p/does-linoleic-acid-induce-obesity-b70

Reading the studies of mice eating salmon grown on pufa show significant effects of the feed.

You have discarded my grass fed beef diet saying it's success is based on exclusion of processed foods, which is partly true, but my exclusion of processed foods began long before and was not enough. In addition to the above, eating from animals with a polystomach (rumen..) where unsaturates are hydrolized into sfa improving the PUFA:SFA ratio close to 1:1 is a huge help.

As in one of my first defenses, I cited cows eating grains and the resultant weight gain, and likely the added weight of the consumer of cows.

With this elimination of nuts, cheeses(from pufa fed(unsaturated protected fats) cows), etc with higher pufa cheeses, has lowered my bs and increased my weight loss while being satiated.

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I didn't claim anything, I quoted the authors of the study that you cited to support your argument. And those authors clearly state that they did not find evidence of insulin resistance. Your convoluted arguments make no sense whatsoever.

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Without a doubt more is known now about all phases of health, direct comparisons are lacking. The general mortality charts of today which are dramatically different than the same as prior to 1900.

I cannot in good conscious imply that physicians were so unobservant that they didn't see morbid obesity in most, nearly universal IR, huge numbers of CVD, CAD, CA and so forth. Too big of an ask.

Jason Fung, noted Egyptians were grains and corn eaters, their agriculture depended on the Nile. They have hieroglyphs of sickles (ate it) vs animals eating it. Also recall their teeth were worn by the sand from the millstones. Their stores of grains were well noted in the bible, years of plenty and famine. I see no proof of 'carnivores'.

Your citation of mention of Angina just after a meal, denotes carbs and not meat. My bs hasn't been over 100 for awhile, even @ 30 mins post meal.

At this point, you may think I am biased. It's undeniable, by my own body. I've checked my bs so many times, my weight. As you have said. Test and Prove, step by step I have and relayed on your blog. I have lost weight on keto, and I have gained weight and bs eating seed oils and carbs, they're hardly inseparable. While avoiding sugar for decades. I have lost weight through elimination of carbs to a point and maintained on keto, yet, bs top of normal, though a GTT, now normal. As previously mentioned switching to a carnivore diet has dropped bs to the bottom of normal range, and I'm losing weight without fasting. I've replied enough regarding the success of my diet.

Benjamin Bikman, Researcher of IR and lipidology and Teacher of Medical students. wrote 2 books. https://cell.byu.edu/directory/benjamin-bikman

While I came by him while listening to hundreds of hours of talks, you like to read, so here is his first book.

https://www.amazon.com/Why-We-Get-Sick-Epidemic/dp/1953295770

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I've engaged with you in a spirit of good faith, but you have not returned the favour.

Firstly, your repetitive use of the straw man fallacy is getting really irksome. At no point did I say that physicians somehow managed to overlook obesity or cancer; I pointed out that they were unable to diagnose myocardial infarction due to lack of diagnostic equipment. Ditto for your perseveration on "seed oils" and sugar. I do not advocate for a diet that contains these ingredients, as I've made abundantly clear in my responses to you, and in my articles.

Secondly, I invited you to provide me with references for your claims. I then provided a detailed critique of that reference (the Paul Mason video), to which you have not responded. I have nointention of wasting my time doing a similarly detailed critique of Jason Fung and his loose relationship with reality, because there are plenty of them already circulating on the web.

Thirdly, I have made it clear that personal anecdote is not persuasive to me. As I wrote in Part 1 of this mini-series, "If a particular diet seems to be working for you, I'm very happy for you - but don't expect me to abandon the recommendations that I currently make, based on over three decades of studying the research literature, and conducting my own clinical practice, because of your n=1 experiment."

I honestly don't know what you're trying to achieve by repeatedly telling me about your personal experience. Do you think that is persuasive? Would you be persuaded to change your diet if I bombarded you with my own personal anecdotes, of how I'm thriving on a totally different diet to you, and how the lives of thousands of my clients have been transformed by this diet? If you wouldn't, ask yourself why that is not persuasive to you, and why you persist with this strategy.

I'm beginning to think that Pirate Studebaker might be right, and that the 'seed oils are the cause of all our ills' canard is a psyop promoted by they/them/those who seek to rule over us. Or maybe it's a religious cult; the people I've encountered who have adopted this ideology certainly behave like religious zealots.

If you are capable of engaging in rational debate that is based on verifiable facts rather than logical fallacies and anecdotes, I will respond to you. Otherwise, I'm not going to waste any more time on this fruitless endeavour.

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From the time I commented on your first post where you questioned my preference for eggs, and animal protein in a low carb setting. And you asked why. To which I responded, as well as the list of your studies only particularly as it pertained to grass fed animal sfa. no matter how many studies have been done to show something safe, there is a worldwide population of people eating both carbs and meat before 'something(s)' beginning on or around 1900, because the first incidence of these is the early 1900. Animal fats were worldwide safe until 1900. You ignored this in your response. Also so were carbs! I can't find a way to lose weight eating them after being harmed by something during the 20th century on wards.

After reading some studies about plaque I got motivated to initiate another modification, too long in steady state. While you worked on these posts, I was busy with required life deadlines. As a self treatment, I switched to Grass fed animal protein, beef/lamb. So far, it's been the easiest diet modification i've tried. I am choosing to only eat when I'm hungry. My attention is more focused than during fasting. I'm now eating less meat than when I was keto. Results. Losing weight and size, so far 8-9#, and lowering bs, and further switching to decaf dropped bs 5-7 points, tested with 2 days of caff then non caff, repeated confirmed.

I'm searching for information, not searching against. There will always be information to make a case with or against, hoping to find truth hiding. It is likely a combination of things, but clearly they are inventions added to our diet, in the past 120 years. These diseases didn't exist before and now they do. For me this is very simple, I have tested and confirmed with my diet.

increased meat, low GI carbs, worked for awhile. bs down. Cutting out seed oils, helped awhile longer. bs down. Fasted to drop weight, but only slight bs change in the morning. Switching to grass fed, helped, bs down, eliminating all plants helping more. Only holdout left, finding grass fed cream and butter.

Novel finding. I went a shoveled snow for 45 mins. came back in bs had gained 18 points with no food. I tested again, 2 hrs later, 10 push ups, and it bumped it up 8 points. No food.

I suppose Gluconeogenesis is now working better than I've ever seen in the past.

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You're commencing your inquiry with a faulty premise, which is that heart attacks (the major cause of death resulting from atherosclerotic heart disease) only emerged in the early 20th century. This is completely untrue.

1) Atherosclerotic heart disease has been identified in Egyptian mummies, e.g. see https://www.sciencedaily.com/releases/2009/11/091117161017.htm. Since only the well-heeled were mummified, and their consumption of animal products was documentably much higher than that of the great mass of the population, there's a plausible link between their animal product consumption and propensity to atherosclerosis.

2) Angina - now known to be a consequence of atherosclerotic heart disease - was described by Heberden in 1768 (see https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2651944/), but its aetiology was unknown. Deaths that occurred as a consequence of atherosclerotic heart disease were not correctly attributed, because...

3) It was simply impossible to correctly identify heart attacks prior to the widespread use of ECGs (first developed in 1902, but not widely used for several decades) and, most particularly, creatine kinase and cardiac troponin assays, which are able to identify even minor heart attacks. And of course, autopsies were rarely performed. Hence, deaths that would now be correctly attributed to heart attack, were misclassified, usually attributed to liver disease or some other acute gastrointestinal complication. In addition, short average lifespans meant that most people died of other causes - especially infections and accidents - before they lived long enough to be vulnerable to a heart attack.

And of course your blood glucose level went up after you exercised on an empty stomach. Exercise stimulates catabolic hormones. This happens in everyone.

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